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. 2023 Nov 22;14:7613. doi: 10.1038/s41467-023-43568-6

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Aggregated expression of disease-associated genes in each cell type (coding region, left; noncoding region, right). b Expression patterns of selected disease-associated genes in each cell type in the ATAC dataset. c Normalized ATAC-seq profiles of SPTBN2 and PLEKHG4 in the cerebellum, with all cell types showing activation of SPTBN2 and PLEKHG4.