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Mitochondrial GpC hypermethylation upregulates nuclear receptors and PPARα target genes involved in bile acid metabolism. Heatmap representation of differentially expressed nuclear receptors (A) or PPARα target genes (B) in three different untreated (none) HepG2 cell lines overexpressing a GpC DNMT MCviPI (MCviPI), a CpG DNMT (MSSSI), or overexpressing a GpC/CpG DNMT deficient MCviPI mutant.
