Table 1.

Specific Conditions for Inflammatory Cardiomyopathy. For all disorders, T2-weighted Images, T2-mapping values and T1-mapping values are abnormal. Further specific findings are listed hereunder for the relevant etiologies

Infectious Etiology
	CMR
Viral myocarditis	• LGE lateral wall or septum (subepicardial or mid-wall) [52, 53] • Low/ normal LVEF
Bacterial myocarditis	• LGE lateral wall (subepicardial or intramural) [54, 55]
Parasitic Myocarditis	• LGE lateral wall (patchy in toxoplasma gondii [56] and midwall, subepicardial, subendocardial or transmural in patients with Trypanosoma cruzi with a prevalence for inferolateral basal and apex) [57, 58]
Non-Infectious Etiology
Eosinophilic myocarditis	• Diffuse subendocardial LGE [59, 60] • Possible association with ventricular thrombus [59]
Systemic Lupus Erythematosus	• Subendocardial, intramyocardial, subepicardial LGE [61, 62]
Systemic Sclerosis	• Prevalent subepicardial, midwall LGE of the septum [63]
Polymyositis	• Subepicardial lateral and inferior wall [64]
Giant Cells Myocarditis	• Subendocardial, and subepicardial diffuse LGE with involvement of both ventricles [65]
Kawasaki Syndrome	• Subendocardial LGE [66]
Sarcoidosis	• Subendocardial, subepicardial, midwall LGE [67]
Chemotherapy	• Subepicardial or mid-wall LGE [68]
Post-Vaccination	• Subepicardial or mid-wall LGE [69, 70]

LGE: late gadolinium enhancement