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. 2023 Nov 25;13:215. doi: 10.1186/s13578-023-01159-4

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Cell cycle distribution analysis of 293T cells treated with small molecule inhibitors. A Analysis of 293T cells unsynchronized (DMSO) and synchronized by DOC (5 μM), NOC (2.5 μM), IRI (10 μM) and MITO (5 μM) for 12 (left panel) and 24 h (right panel) showing DOC and NOC arrested cells at G2/M stage and IRI and MITO increased the proportions at both S and G2/M phases of cell cycle. B Quantification of cell cycle distribution by measuring the area representing the specific cell cycle stage in FACS histogram. The mean values and error bars (SD) were calculated from three experiments