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. 2023 Nov 27;18:80. doi: 10.1186/s13062-023-00439-w

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — RCC2 drives cell movement and EMT of prostate cancer. A, B Representative images and statistics chart of cell movement in DU145 and PC3 cells transfected with pcDNA-RCC2 and pcDNA-ctrl or siRCC2#1, siRCC2#2, and siCtrl for 24 h were evaluated using the Transwell assay. C, D Representative images and statistics chart of cell invasion of DU145 and PC3 cells. E, F, G, H A wound healing assay was adopted to measure cell migration ability. I, J The expression levels of EMT markers, including Snail, E-cad and Vim, in DU145 and PC3 cells. **P＜0.01, ***P＜0.001