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[Preprint]. 2023 Nov 16:2023.11.14.567084. [Version 1] doi: 10.1101/2023.11.14.567084

Fig 4. Two distinct Septin complexes are functioning during cell wound repair.

Fig 4.

(A-I) Confocal max projection images of wounds generated in embryos expressing Sep2-GFP (A, D, G), Sep5-RFP (B, E, H), or ChFP-Pnut (C, F, I) in wildtype (A-C), Sep1 RNAi knockdown (D-F), or Sep4 RNAi knockdown (G-I) backgrounds. Scale bar: 20 µm. (J-L) Septin recruitment (area under the curve of fluorescence profile) for Sep2-GFP (J), Sep4-GFP (K), or ChFP-Pnut (L) in control (wildtype), Sep1 RNAi, and Sep4 RNAi backgrounds at 70% wound closure (100% = fully open). Error bars represent ±SEM. Kruskal-Wallis test was performed for J-L. Comparison to control is indicated by blue asterisks: * p<0.05, ** p<0.01, *** p<0.001, **** p<0.0001, ns is not significant.