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. 2023 Nov 30;25:72. doi: 10.1186/s12968-023-00986-1

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Non-ringlike LGE and corresponding electrocardiograph. A 17-year-old woman had a prior history of syncope for 1 year with genetically diagnosed LD- ACM (PKP2 mutation). A LGE-CMR images showed the multifocal subepicardial fibrosis with a non-ringlike pattern involving the LV free wall and septum in both the short axis (left panel: from base to apex) and the long axis view (right panel: from 2 chamber to 4 chamber) (white arrowheads). B The 12-lead electrocardiograph showed multifocal PVC. LGE late gadolinium enhancement, LD-ACM left dominant arrhythmogenic cardiomyopathy, PKP2 plakophilin-2, CMR cardiac magnetic resonance, LV left ventricular, PVC premature ventricular contraction