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. 2023 Nov 13;26(12):2104–2121. doi: 10.1038/s41593-023-01480-8

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 10 — Principal component analysis plot for hippocampal clusters 1, 6, 7, 9, 28, astrocyte subclusters 3, 5, 7, and microglia subclusters 2, 8, 11 against all measured pathologies (hippocampal volume and coverage areas of AT8, Iba1, CD68, GFAP, and S100β). PC1 and PC2 showed the overall relationship between clusters based on similarity of the estimated log odds ratio per unit change in six pathologies per cluster/subcluster. AS, astrocyte; MG, microglia. For details of the analyses, see Methods.