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. 2023 Dec 1;18:82. doi: 10.1186/s13062-023-00435-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — uc.291, ACTL6A, FLG and LOR expression in an in vitro psoriasis-like model. A Analysis of uc.291 and ACTL6A in human differentiated keratinocytes not treated (NT) and treated with IL-22 was determined by RT-qPCR (n = 3). B Bioinformatics analysis of ACTL6A in untreated human epidermal keratinocytes (UNT) (n = 3) and treated with IL-22 (n = 3) (GSE7216). C Analysis of FLG and LOR in human differentiated keratinocytes not treated (NT) and treated with IL-22, determined by RT-qPCR (n = 3). The p-value was obtained using t-test student; p < 0.05; n.s. not significant. D Bioinformatic analysis of FLG and LOR on Untreated (UNT) (n = 3) and IL-22 treated human epidermal keratinocytes (n = 3) (GSE7216). For all experiments and datasets analysis of data sets, the p-value was obtained using a t-test student; p < 0.05; n.s. not significant. Replicae are biological replicates