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. 2023 Dec 6;15:104. doi: 10.1186/s13073-023-01262-8

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — BRCA1 methylation in matched blood and tumor samples in breast cancer patients. A Molecular and histological characteristics (rows) of all samples (N = 55; columns) belonging to matched sample pairs carrying BRCA1 methylation in the blood (WBC) and/or tumor. TNBC, triple-negative breast cancer; HER2, Human epithelial-like receptor-2; ER, estrogen receptor; basal, basal-like gene expression profile; Her2, HER2-enriched gene expression profile; LumA, luminal A gene expression profile; LumB, luminal B gene expression profile; Normal, normal-like gene expression profile; IDC, invasive ductal carcinoma; ILC, invasive lobular carcinoma. B Concordance of BRCA1-methylation status in WBC and tumor tissue among all patients analyzed, stratified for tumors belonging to the different breast cancer subgroups