A–D
Chronically reactivating hM3D‐labelled PV neurons did not affect the depressive‐like behaviours, as evaluated by SPT (A, subgroups, F
(1, 20) = 0.8020, P = 0.3812; treatment, F
(2, 40) = 2.940, P = 0.0644; subgroups × treatment, F
(2, 40) = 0.03915, P = 0.9616), FUST (B, subgroups, F
(1, 20) = 0.04271, P = 0.8384; treatment, F
(1, 20) = 2.907, P = 0.1037; subgroups × treatment, F
(1, 20) = 0.007554, P = 0.9316) and FST (C, two‐tailed unpaired t‐test, t
(20) = 0.4940, P = 0.6267) and the locomotor activity (D, two‐tailed unpaired t‐test, t
(20) = 0.2734, P = 0.7874).
E, F
Chronically reactivating hM3D‐labelled PV neurons did not induce insulin resistance evaluated by GTT (E, left, fasting blood glucose level, two‐tailed unpaired t‐test, t
(20) = 0.4146, P = 0.6829; middle, glucose tolerance test, subgroups, F
(1, 20) = 0.2836, P = 0.6002; time, F
(4, 80) = 497.7, P < 0.0001; subgroups × treatment, F
(4, 80) = 0.7470, P = 0.5629; right, the area under the curve of GTT, two‐tailed unpaired t‐test, t
(20) = 0.7050, P = 0.4889) and ITT (F, left, subgroups, F
(1, 20) = 0.8558, P = 0.3659; time, F
(5, 100) = 131.8, P < 0.0001; subgroups × treatment, F
(5, 100) = 1.249, P = 0.2923; right, the area under the curve of ITT, two‐tailed unpaired t‐test with Welch's correction, t
(20) = 0.5915, P = 0.5608).