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. 2023 Dec 10;8:449. doi: 10.1038/s41392-023-01720-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — Epigenetic and posttranslational modification of ferroptosis in Kidney disease. AKI Acute kidney injury, ALK activin receptor-like kinase, CHAC1 Cation transport regulator-like protein 1, DNMT1 DNA methyltransferase 1, FMR1 FMRP translational regulator 1, IRI ischemia/reperfusion injury, SAKI sepsis-induced acute kidney injury, TBK1 TANK-binding kinase 1, TIGAR TP53-induced glycolysis and apoptosis regulator