Fig. 6.

Acquired injury in the normally formed brain. (i) Diffuse left sided white matter and cortical parieto-occipital infarction (red arrows). High T2 signal is also seen within the left posterior thalamus, suggestive of secondary network injury (red arrowhead). A video of this study is available as an Online Supplement. (ii) Bilateral basal ganglia, thalamic, cortico-spinal tract, and mesencephalon injury secondary to perinatal hypoxia-ischaemia. Hypoxic-ischaemic injury revealed as abnormal low T2 within the deep grey matter (Ad), cerebral peduncles (Ac), hippocampi (Ah) and brainstem (Ab). There is diffuse abnormal high T2 signal in the white matter (C), with areas of sub-cortical white matter highlighting on 64mT T2w images (D). The Posterior Limb of the Internal Capsule is abnormally T2 hyperintense -abnormal for gestational maturity (E). Additional findings include widened interhemispheric fissure (F), and bilateral sub-ependymal cysts (G).