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. 2023 Dec 4;14:1276951. doi: 10.3389/fmicb.2023.1276951

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Copyright © 2023 Yang, Wang, Jiang, Zhang, Du, Li and Liu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The host proteins identified from gallstone and polyp bile samples. (A) Gene ontology (GO) enrichment was performed on the upregulated human proteins in CHL compared to polyp using the g:Profiler tool (https://biit.cs.ut.ee/gprofiler/gost) (Raudvere et al., 2019). (B) Differential expression of complement proteins between CHL and polyp samples. p values were calculated using unpaired Welch’s t test. (C) Two mucin proteins were upregulated in CHL compared to polyp. (D) GO enrichment of the downregulated human proteins in CHL compared to polyp. GO_MF, Gene ontology of molecular function; GO_CC, cellular component; GO_BP biological process; REAC, Reactome; WP, WikiPathways. (E) Proteins associated with bile secretion were downregulated in CHL compared to polyp.