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. 2023 Dec 18;14:8399. doi: 10.1038/s41467-023-44197-9

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — During M. oryzae infection, MoSPAB1 was secreted from invasive hyphae and translocated to rice cytoplasm and the host nucleus. MoSPAB1 binds to the (C/A)(G/C)(C/G)T(T/C)G(C/A)(T/C) motif on the Bsr-d1 promoter and activates Bsr-d1. BSR-D1 accumulation accelerates H₂O₂ degradation by inducing expression of peroxidase genes, increasing disease susceptibility. Rice uses MYBS1 to suppress Bsr-d1 expression in order to combat M. oryzae MoSPAB1. Deletion of MoSPAB1 reduces M. oryzae pathogenicity on rice.