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. 2023 Oct 24;28(6):921–933. doi: 10.1007/s12192-023-01386-1

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — EGCG and salidroside suppressed HH-induced myocardial apoptosis and regulated the expression of antioxidant-related proteins Nrf2 and HO-1. a Protein levels of Nrf2, HO-1, Bax, caspase-3, and Bcl-2 in the LV tissues were measured by immunoblotting. The band intensity of b Bax, c caspase-3, d Bcl-2, e Nrf2, and f HO-1 was quantified. Nrf2, nuclear factor E2-related factor 2; HO-1, heme oxygenase-1; LV, left ventricle. **P < 0.01 and ***P < 0.001 compared with the P group; #P < 0.05, ##P < 0.01, and ###P < 0.001 compared with the HH group