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. 2023 Dec 11;14:1224383. doi: 10.3389/fimmu.2023.1224383

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Copyright © 2023 Nascimento, Huot-Marchand, Fanny, Straube, Le Bert, Savigny, Apetoh, Van Snick, Trovero, Chamaillard, Quesniaux, Ryffel, Gosset, Gombault, Riteau, Sokol and Couillin

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Transfer of impaired cigarette smoke-induced airway inflammation of Nlrp6^-/- mice to wild type mice by cohousing. (A) WT mice and Nlrp6^-/- mice were single-housed (SH) or co-housed (CH) for 12 weeks and exposed for 4 days of CS. BALF, lung and caecum were collected 16 hours after the last exposure. Principal components analysis (PCA) analysis of WT and Nlrp6^-/- SH or CH in (B) air or (C) CS conditions, (D) WT SH in air and CS conditions, and (E) Nlrp6^-/- SH in air and CS conditions. (F) BALF total cells and (G) neutrophils. (H) BALF CXCL5 and (I) CXCL1 levels. Data are representative of tywo expirements and are expressed as mean values ± SEM and n= 6-9 mice per group, ns, non significant, group), *p < 0.05, **p < 0.01, ***p < 0.001, ****P < 0.0001.