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Proposed model of the NP–Z interaction during the viral life cycle. At a late stage of infection, the cellular concentration of Z is high favoring for NP–Z interaction. This leads to recruitment of RNPs to the cell membrane by myristoylated Z for virion assembly (left). During virus entry into the host cell via the endosomal pathway, the lower pH leads to the dissociation of NP from Z, which coats the inner virion membrane. This leads to the release of RNPs into the cytosol.
