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. 2024 Jan 3;19:2. doi: 10.1186/s13018-023-04475-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Chondrogenic differentiation and activity regulation of BMSCs. A. LINC00665 level in BMSCs chondrogenic differentiation within 14 days. B. The change in cartilage matrix proteoglycan GAG level within 14 days of BMSCs chondrogenic differentiation was detected by Alcian blue staining. (** P < 0.01, *** P < 0.001, compared with no differentiation group) C. Transfection efficiency of LINC00665 overexpression in BMSCs. D. LINC00665 overexpression inhibited the proliferation of BMSCs. E. LINC00665 overexpression increased the apoptosis rate of BMSCs. F. LINC00665 overexpression reduced the chondrogenic differentiation of BMSCs. (***P < 0.001, compared with the control group)