Skip to main content
. 2023 Apr 25;202(1):122–132. doi: 10.1007/s12011-023-03676-2

Table 2.

Univariate analysis of predictive factors for ATO-induced hepatotoxicity

Clinical characteristics All Patients (N = 122)
Cases Controls
Hepatotoxicity, no. (%) Nonhepatotoxicity, no. (%) P
Total 112 (91.80%) 10 (8.20%)
Age, years
   > 50 36 (92.31%) 3 (7.69%) 0.889a
   ≤ 50 76 (91.57%) 7 (8.43%)
Sex
  Female 53 (92.98%) 4 (7.02%) 0.749a
  Male 59 (90.77%) 6 (9.23%)
WBC count, × 109/L
   ≤ 10 90 (92.78%) 7 (7.22%) 0.426a
   > 10 22 (88%) 3 (12%)
Platelet count, × 109/L
   ≥ 30 44 (89.80%) 5 (10.20%) 0.508
   < 30 68 (93.15%) 5 (6.85%)
Hemoglobin level, g/L
   ≥ 80 58(95.08%) 3 (4.92%) 0.323a
   < 80 54 (88.52%) 7 (11.48%)
Fibrinogen level, g/L
   ≥ 1 75 (93.75%) 5 (6.25%) 0.279
   < 1 37 (88.10%) 5 (11.90%)
Prophylactic hepatoprotective agents
  Yes 22 (78.57%) 6 (21.43%) 0.01**a
  No 90 (95.74%) 4 (4.26%)
Anti-leukocytosis agents
  Single ATO 68 (89.47%) 8 (10.53%) 0.317a
  ATO + chemotherapy 44 (95.65%) 2 (4.35%)

Bold values and * are statistically significant (P < 0.05); Bold values and * are statistically significant (P < 0.01); ATO, arsenic trioxide; a, P-values for categorical variables are from Fisher’s exact test (n < 5)