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. 2023 Dec 27;14:1291965. doi: 10.3389/fphar.2023.1291965

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Copyright © 2023 Abdeen, Elsabagh, Elbasuni, Said, Abdelkader, El-Far, Ibrahim, Mihaela, Fericean, Abdelfattah, El-Hewaity, Elbarbary, Kadah and Ibrahim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The protective effect of ChV against AFT-induced kidney injury is underpinned by molecular processes. AFTs, aflatoxins; ChV, Chlorella vulgaris; GCLC, glutamate-cysteine ligase catalytic subunit; GPx, glutathione peroxidase; GR, glutathione reductase; GSH synthetase, glutathione synthetase; IL1RAP, interleukin-1 receptor accessory protein; IL6RA, interleukin-6 receptor subunit alpha; MDA, malondialdehyde; ROS, reactive oxygen species; SOD, superoxide dismutase; TNFR, tumor necrosis factor receptor; TRAF1, tumor necrosis factor receptor-associated factor 1.