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. 2014 Dec 18;2014(12):CD008146. doi: 10.1002/14651858.CD008146.pub2

Micallef 2009.

Methods Block‐randomised, double‐blind, placebo‐controlled, multicentre trial
Duration: 12 months
Participants Participants were recruited from 3 hospital sites in France
195 patients were initially screened and 179 (110 women and 69 men) with a clinical diagnosis of CMT1A confirmed by genotyping with duplication in 17p11.2 were subsequently randomised prior to intervention
Participants were aged between 18 and 70 years of age and had to have at least one motor symptom or sign (gait disorder, distal amyotrophy, foot deformation or muscle weakness). People with any cause of neuropathy other than CMT1A and other medical conditions: kidney stones, hypersensitivity or intolerance to ascorbic acid or lactose, uncorrected oxalosis, cardiovascular disease, poliomyelitis or radiotherapy, those for whom ascorbic acid supplementation was contra‐indicated, women of childbearing age who were pregnant, breastfeeding or not using adequate contraception, were excluded
Interventions Participants were randomised in a 1:1:1 ratio to ascorbic acid 1 g, (n = 56), ascorbic acid 3 g (n = 61) or placebo containing lactose (n = 62), all given as a single daily administration of 3 hard gelatine capsules for 12 months.
Outcomes Primary outcome: Charcot‐Marie‐Tooth neuropathy score (CMTNS) at 12 months after commencement of intervention
Secondary outcomes
  1. Muscle strength ‐ QMT

  2. Gait velocity ‐ timed 10 metre walk

  3. Activity ‐ ODSS

  4. Quality of life ‐ Medical Outcomes SF‐36

  5. VAS for fatigue, pain, cramps and difficulty walking

  6. CGI‐S

Funding No indication of funding given
Declarations of interest One study author (MF) is one of the inventors of the patent for ascorbic acid for the treatment of CMT1A licensed to Murigenetics. Two of the study authors (MF and OB) are scientific advisers to Murigenetics and are 15% shareholders in the company.
Notes Participants were recruited from Sept 2005 to September 2007
Risk of bias
Bias Authors' judgement Support for judgement
Random sequence generation (selection bias) Low risk Block randomisation stratified to site and sex for subgroup analyses with sequence generation using an independent contract research organisation
Allocation concealment (selection bias) Low risk Randomisation list sent to an independent research organisation for preparation of ascorbic acid and placebo capsule (identical hard gelatine capsules)
Blinding (performance bias and detection bias) 
 All outcomes Low risk Ascorbic acid and placebo formulated from hard gelatine capsules (identical in weight, appearance and taste). Placebo contained lactose
Incomplete outcome data (attrition bias) 
 All outcomes Low risk Analyses were on an intention‐to‐treat basis. Reporting of dropouts (total 16) was effective: 6 withdrew (1 in placebo, 4 in 1g ascorbic acid and 1 in 3 g ascorbic acid group) due to adverse events (cystitis, myalgia, gastralgia, depression, colitis and insomnia), 2 were lost to follow up (1 in placebo and 1 in 3 g ascorbic acid group), 6 withdrew consent (1 in placebo, 4 in 1 g ascorbic acid and 1 in 3 g ascorbic acid), 1 was non‐compliant with treatment in the placebo group and 1 placebo participant was missing a baseline CMTNS score
Selective reporting (reporting bias) Low risk All stated primary and secondary outcomes were fully reported
Other bias Unclear risk Intention‐to‐treat analysis meant sufficient sample size for adequate power. No indication of how missing data were dealt with for intention‐to‐treat analyses. Therefore difficult to determine risk of bias due to method