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. 2023 Nov 22;6(4):805–827. doi: 10.20517/cdr.2023.77

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© The Author(s) 2023.

© The Author(s) 2023. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, sharing, adaptation, distribution and reproduction in any medium or format, for any purpose, even commercially, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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(A) Schematic illustration of a unimolecular micelle for inhibiting pump-mediated drug resistance; Cell viability of (B) HepG2/MDR1 cells and (C) HeLa/MDR1 cells treated with different doses of DOX, β-CD-g-PLC-SS-PEG/DOX, and β-CD-g-PLC-SS-PEG-FA/DOX. This figure is quoted with permission from Li et al.^[89]. CD: Cyclodextrin; DOX: doxorubicin; FA: folic acid; GSH: glutathione; MDR1: multidrug resistance gene; PEG: poly (ethylene glycol).