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. 2023 Oct 13;30(1):e14486. doi: 10.1111/cns.14486

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© 2023 The Authors. CNS Neuroscience & Therapeutics published by John Wiley & Sons Ltd.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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ANA‐12 treatment reversed the protective effects of DEX in alleviating long‐term cognitive impairment following HIBD. (A) Representative swimming tracks of the rats from the place navigation test on day 5 and the probe test on day 6. (B) Average escape latency to reach the hidden platform during the place navigation test over days 1–5. (C) Number of platform crossings during the probe test. (D) Time spent in the target quadrant during the probe test. (E) Swimming speeds of the rats during the probe test. DEX, dexmedetomidine; HI, hypoxic—ischemia; HIBD, hypoxic‐–ischemic brain damage. Data were expressed as the mean ± SD (n = 10 per group); *p < 0.05 vs. the sham group; ^# p < 0.05 vs. the HI group; ^& p < 0.05 vs. the HI + DEX group.