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. 2024 Jan 10;10(1):e003901. doi: 10.1136/rmdopen-2023-003901

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© Author(s) (or their employer(s)) 2024. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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(A) Differential expression genes in intestinal specimens of patients with NXP2⁺DM compared with healthy control. (B) Interferon type I genes upregulated in the intestinal of patients with NXP2⁺DM. (C) Upregulated and downregulated pathways indicated by functional enrichment analysis of differentially expressed genes. (D) Enrichment scores of metabolic genes revealed various metabolisms were inhibited in patients with NXP2+DM. (E) Gene sets enrichment analysis showed epithelial mesenchymal transition, focal adhesion and TNF-α signalling were upregulated while oxidative phosphorylation was downregulated. DM, dermatomyositis; NXP2, nuclear matrix protein 2. TNF, tumor necrosis factor. FDR, false discovery rate. NES, normalized enrichment score.