Figure 4. C1ql3 regulates cAMP signaling and amplifies GSIS in β-cells.
Upon secretion from β-cells, C1ql3 signals in an autocrine manner by binding to its receptor BAI3 to regulate insulin secretion by inhibiting cAMP signaling.
Upon secretion from β-cells, C1ql3 signals in an autocrine manner by binding to its receptor BAI3 to regulate insulin secretion by inhibiting cAMP signaling.