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. 2024 Jan 11;25(2):905. doi: 10.3390/ijms25020905

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© 2024 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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The expression of m⁶A writers in HuLM cells in response to I-BRD9 treatments and the experimental model. The RNA expression of METTL3 (A) in HuLM cells treated with I-BRD9. (B) The comparison of protein levels of m⁶A readers (YTHDC1 and YTHDF2) in vehicle- and I-BRD9-treated HuLM cells. (C) The experimental model shows that I-BRD9 treatment promotes apoptosis, induces cell cycle arrest, represses ECM accumulation, and reprograms the epigenome and epitranscriptome in UF cells. Figure 8C was created using BioRender software (BioRender.com, accessed on 23 December 2023). **** p < 0.0001.