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. 2024 Jan 26;7:131. doi: 10.1038/s42003-024-05826-1

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a The number and strength of cell interactions among the major cell types of ovarian cancer. b The UMAP plot shows the fibroblast sub-clusters in ovarian tumor and metastatic tumor foci. c The UMAP plot color coded for the expression of marker genes for the fibroblast subtypes. d The bar chart shows the GO enrichment term of marker genes in Fib8. e The dot plot shows variation of ECM gene expression in myCAF with stage. f The violin plot shows change of ECM score in myCAF with stage (Total 7,438 single cell). *P < 0.05; **P < 0.01; ***P < 0.001, ****P < 0.0001. g Kaplan–Meier OS curves of patients with TCGA HGSOC grouped by the top10 marker gene of myCAF. P values were calculated by a log rank test.