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. 2024 Jan 29;24:140. doi: 10.1186/s12879-024-08973-2

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Analysis of selected Gene Ontology (GO) terms. A Heatmap showing the top 10 differentially expressed genes (DEGs) (based on FDR) enriched in humoral responses, cytokine signaling, TNF production and T cell activation GO terms. The genes (rows) are grouped by the clinical phenotype: A = Asymptomatic, U = Uninfected, FU = Febrile malaria ensuing from Uninfected, FA = Febrile malaria ensuing from Asymptomatic. B Boxplots showing the normalized expression levels of selected DEGs per GO term. The boxplots are labelled based on the clinical phenotype: A = Asymptomatic, U = Uninfected, FU = Febrile malaria ensuing from U and FA = febrile malaria ensuing from A. Pairwise statistical analyses indicated in the plots are Wilcoxon tests corrected for multiple testing (Benjamini & Hochberg, * = < 0.05, ** = < 0.01 and *** = < 0.001)