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. 2024 Jan 30;21:36. doi: 10.1186/s12974-024-03028-4

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Meningeal lymphatic function in aged mice is more vulnerable to sepsis. A Representative immunofluorescence images of OVA-647 accumulation in dCLNs. B Representative immunofluorescence images of meningeal whole-mounts of OVA-647 stained with DAPI and Lyve-1. C Graph depicting the percentage area of OVA-647 coverage in the dCLNs. D Graph depicting the percentage area of OVA-647 coverage in the meningeal lymphatic vasculature. E Graph depicting the percentage area of Lyve-1 coverage in the meningeal lymphatic vasculature. n = 5. **P < 0.01, aged control group vs. aged + LPS group, ^# P < 0.05, young + LPS group vs. aged + LPS group. All data are expressed as the mean ± SD