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. 2024 Jan 31;24(1):29. doi: 10.1007/s10238-023-01260-5

Table 2.

lncRNA's functions and related signaling pathways in GC and CRC

Cancer LncRNA Regulation Signaling pathways Function References
CRC PINT Down` PRC2 Inhibits proliferation [186]
MALAT1 Up miR-129-5p, HMGB1, miR-663a Improve proliferation, migration and invasion [187]
Loc285194 Down miR-211 Inhibits proliferation [188]
Linc02418 Up miR-34b-5p, Bcl-2 Stimulates growth, mobility, invasion, apoptosis inhibition [189]
Linc00261 Down β-catenin, Wnt Inhibits proliferation, apoptosis, invasion, migration, and drug resistance [190]
Linc01567 Up miR-93 Stimulates proliferation, invasion, migration [191]
H19 Up miR138, HMGA1, miR-675-5p Improve invasion, migration, and drug resistance [119]
HOTAIR Up E-cadherin, vimentin, MMP-9 Increases migration, invasion [192]
GSEC Up DHX36 Elevates migration [193]
FAM83H-AS1 Up TGF-β signaling Elevates tumorigenesis [194]
HNF1A-AS1 Up miR-34a/p53 Enhances proliferation, migration, invasion [195]
HULC Up miR-613, RTKN, vimentin, N-cadherin, E-cadherin Elevates proliferation, migration, invasion [196]
FAL1 Up STAT3, TGF-β1, Bcl-2, p65, PCNA Enhances proliferation, invasion, inhibits apoptosis [197]
DACOR1 Down Cystathionine β-synthase Inhibits proliferation, increases DNA methylation [198]
CCAT1 Up c-Myc Promotes proliferation, invasion, drug resistance [199]
CCAT2 Up miR-145, WNT Enhances growth, metastasis [199]
CASC15 Up miR-4310, LGR5, Wnt/β-catenin Promotes proliferation, migration, invasion [200]
CASC19 Up N/A Increases migration [201]
ATB Up E-cadherin, ZO-1, ZEB1, N-cadherin Enhances invasion, induces EMT [202]
PVT1 Up miR-26b, miR-30d-5p/RUNX2 Elevates proliferation, migration, invasion [203]
ROR Up miR-145 Enhances proliferation, migration, invasion [204]
TUG1 Up miR-26a-5p, MMP-14, VEGF, MAPK, Hsp27 Promotes proliferation, migration, invasion, EMT, inhibits apoptosis [205]
UPAT Up UHRF1 Elevates survival [206]
XIST Up miR-34a, Wnt, β-catenin cyclin D1, c-Myc, MMP-7 Promotes proliferation [207]
ZEB1-AS1 Up miR-455-3p, PAK2 Enhances proliferation, migration, invasion [208]
ZFAS1 Up ZEB1, E-cadherin, ZO-1, vimentin, N-cadherin Increases proliferation, invasion, EMT, inhibits apoptosis, [209]
SNHG1 Up Wnt/β-catenin, c-Myc, cyclin D Increases proliferation, invasion, migration, inhibits apoptosis [210]
SNHG7 Up miR-193b, K-ras/ERK/cyclinD1 Increases proliferation, inhibits apoptosis [211]
SNHG15 Up Slug Enhances proliferation, migration [212]
SNHG17 Up miR-375, CBX3 Elevates proliferation, migration, invasion [213]
Linc01106 Up miR-449b-5p, Gli Confers proliferation, migration, stemness [214]
Linc01234 Up miR-642a-5p, SHMT2 Promotes proliferation [215]
Linc00657 Down CAPN7, PI3K/Akt Inhibits proliferation, invasion, induces apoptosis [216]
Linc01578 Up NF-κB, YY1 Enhances metastasis [217]
CYTOR Up β-catenin/TCF complex Promotes migration, invasion, EMT [218]
BC200 Up STAT3, β-catenin Increases proliferation, invasion, EMT, apoptosis inhibition, cell cycle regulation [219]
LincDUSP Up ATR, p53, E2F, c-Myc Promotes proliferation, stem cells, modulates DNA damage response and cell cycle, inhibits apoptosis [220]
USP2-AS1 Up Phosph-YAP Enhances proliferation, metastasis [221]
LINC00152 Up MAPK LINC00152 suppression significantly reduces the expression of p-ERK-1/2, p-MEK1/2, and c-fos without involving the ERK-1/2 and MEK1/2 expressions. SA treatment changes the function of LINC00152 [222]
PICART1 Down MAPK The upregulation of PICART1 reduces the pERK expression, whereas PICART1 silencing reverses the expression of p-ERK [223]
AOC4P Up MAPK The suppression of AOC4P reduces the expression of ERK1, JNK, and p38 [224]
GC linc00483 Up MAPK The suppression of Linc00483 reduces the expression of c-Jun without affecting the p-Jnk, p53, and p-p38 expression [225]
CRNDE Up PI3K/AKT The repression of CRNDE significantly downregulates pPI3K and p-Akt expression [226]
LINC01559 Up PI3K/AKT The suppression of LINC01559 reduces the expression of PGK1, p-PI3K, p-AKT, and mTOR. IGF-1 treatment (PI3K activator) significantly reverses the LINC01559 knockdown induced phosphorylation of PI3K, AKT, and mTOR [227]
PICART1 Down PI3K/AKT The upregulation of PICART1 reduces p-AKT expression, whereas PICART1 inhibition increases p-AKT expression [223]
LINC01419 UP PI3K/AKT The suppression of LINC01419 in GC cells reduces the expression of p-AKT1 and p-mTOR but does not affect their total levels [228]
LOC101928316 Down PI3K/AKT LOC101928316 upregulation decreases the expression levels of PI3K, p-AKT, mTOR, and p-mTOR. However, the knockdown of LOC101928316 upregulates AKT3, mTOR, and p-mTOR expression and suppresses PTEN expression [229]
PCAT18 Down PI3K/AKT PCAT18 inhibits the PTEN/PI3K/AKT signaling pathway by sponging miR-107, leading to the inhibition of GC progression [230]
OGFRP1 UP PI3K/AKT The silence of OGFRP1 downregulates the expression of p-AKT, leading to the suppression of cell-cycle progression and induction of apoptosis [231]
ADAMTS9-AS2 Down PI3K/AKT The inhibition of ADAMTS9-AS2 increases the expression of p-PI3K and p-AKT. The administration of the PI3K inhibitor LY294002 reverses the negative effect of ADAMTS9-AS2 [232]
FOXD1-AS1 UP PI3K/AKT FOXD1-AS1 activates the PI3K/AKT/mTOR pathway via the upregulation of PIK3CA, leading to an aggravation of GC progression and chemoresistance [233]
TMPO-AS1 UP PI3K/AKT The silence of TMPO-AS1 inhibits the PI3K/ AKT/mTOR signaling pathway by downregulating the expression of BRCC3 via releasing miR-126-5p [234]
XLOC_006753 UP PI3K/AKT X LOC_006753 knockdown reduces the expression of PI3K, p-AKT (Thr308), p-AKT (Ser473), and p-mTOR (Ser2448), causing to the enhance of MDR [235]
MIR4435-2HG UP Wnt/ β-catenin The suppression of MIR4435-2HG reduces β-catenin expression in GC xenografts and inhibits the trans-activating activity of β-catenin [236]
GASL1 DOWN Wnt/ β-catenin The upregulation of GASL1 reduces β-catenin expression in GC cells, while GASL1 knockdown increases β-catenin expression. The administration of a Wnt agonist reduces the negative function of GASL1 on GC cell [237]
LINC01314 DOWN Wnt/ β-catenin The overexpression of LINC01314 downregulates the expression of Wnt-1, β -catenin, cyclin D1, and N-cadherin, while it upregulates E-cadherin expression [238]
FAM83H-AS1 UP Wnt/ β-catenin Silencing FAM83H-AS1 decreases the expression of β-catenin [239]
ZEB2-AS1 UP Wnt/ β-catenin ZEB2-AS1 activates the Wnt/ β-catenin signaling pathway by upregulating ZEB2 expression in GC cell [240]
LINC00665 UP Wnt/ β-catenin LINC00665 knockdown decreases the expression of β-catenin and cyclin D1 in GC cells, whereas it increases GSK-3b expression [241]
TOB1-AS1 DOWN Wnt/ β-catenin The knockdown of TOB1-AS1 increases the expression of β-catenin, c-Myc, cyclin D1, and N-cadherin [241]
lincRNA-p21 DOWN Wnt/ β-catenin The upregulation of lincRNA-p21 reduces the expression of β-catenin and c-Myc [242]
GATA6-AS1 DOWN Wnt/ β-catenin The upregulation of GATA6-AS1 downregulates β-catenin levels and decreases intranuclear β -catenin expression. In GATA6-AS1-silencedGC cells treated with LiCl, β-catenin expression is upregulated [243]
HCG11 UP Wnt/ β-catenin The suppression of HCG11 inhibits the proliferation of GC cells by inhibiting the activity of the Wnt signaling pathway. The administration of LiCl the effect of HCG11 silence on the proliferation [244]
LINC01133 DOWN Wnt/ β-catenin LINC01133 suppresses the nuclear accumulation of β-catenin in GC cells by sponging miR-106a-3p and promoting APC expression [245]
LINC01503 UP Wnt/ β-catenin Silencing LINC01503 in GC cells reduces the expression of β-catenin, cyclin D1, and c-Myc, whereas LINC01503 upregulation reverses their expression [246]
LINC01225 UP Wnt/ β-catenin LINC01225 knockdown reduces the expression ofWnt1and β-catenin in GC cells, whereas it does not affect the expression or Ser9 phosphorylation of GSK-3b [247]
BANCR UP NF-kB BANCR silence decreases the expression of NFkB1 (P50/105) and inhibits the activity of NF-kB1 30 UTR [248]
LINC01410 N/A NF-kB The upregulation of LINC01410 enhances the nuclear signals of NF-kBp65 and the expression of p-IKK-b, p-IkBa, and c-FLIP in GC cells, whereas LINC01410 reverses their expression [249]
KRT19P3 DOWN NF-kB KRT19P3 inactivates the NF-kB signaling pathway by promoting the degradation of IkBa induced by COPS7A suppression [250]
ANRIL N/A NF-kB The inhibition of ANRIL in GC cells reduces the proteinlevelsofp65in the nucleus and the mRNA levels of NF-kB target genes [251]
ASB16-AS1 UP NF-kB ASB16-AS1 activates the NF-kB signaling pathway by upregulating TRIM37 expression [252]
NALT1 UP NOTCH NALT1 suppression reduces the expression of NOTCH1, NICD, and the downstream target genes of the notch signaling pathway, consisting of HES1 and HES5 [253]
MIR22HG DOWN NOTCH The suppression of MIR22HG enhances the expression of HEY1 and nucleus NOTCH2 [254]
EGOT UP Hedgehog The inhibition of EGOT reduces the expression of Shh, SUFU, and Gli1 at both the transcription and protein levels [255]
HOXD-AS1 UP STAT3 The suppression HOXD-AS1 downregulates the expression of p-JAK2 and p-STAT3 [256]
SNHG16 UP STAT3 The knockdown of SNHG16 in GC cells reduces the expression of JAK2 and p-STAT3 by sponging miR-135a [257]
NEAT1 UP STAT3 Silencing NEAT1 decreases STAT3 expression by sponging miR-506 [258]
PVT1 UP STAT3 PVT1 upregulation enhances the accumulation of p-STAT3 in the nucleus by inhibiting its ubiquitin-dependent degradation and increases the transcriptional activity of STAT3 [259]
CTC-497E21.4 UP RhoA The suppression of CTC-497E21.4 regulates the expression of total and active RhoA, CDC42, and Rac1 in GC cells [260]
NORAD UP RhoA Silencing NORAD in GC cells decreases the expression of RhoA and ROCK1 [261]
HOTAIR UP RhoA The upregulation of HOTAIR increases the expression of CXCR4, RhoGEF, PI3K, ROCK, PAK, and PKN (262)
UP STAT3 Silencing HOTAIR reduces the expression of STAT3 and cyclin D1