Table 1.

Clinical and laboratory features of 13 cases of feline hyperaldosteronism^*

Case no.	Breed	Age (yrs)	Sex	Clinical presentation	K	Na	CK	Urea	Creat	TT4	U SG	BP (mmHg)	PAC (pmol/l)
1	DSH	13	FN	Blindness. Bilateral retinal detachment. No evidence of polymyopathy.	↓	N	N	↑	N	N	1.034	200	877
2	DSH	13	FN	Blindness. Bilateral retinal detachment. No evidence of polymyopathy.	↓	N	nd	N	N	N	nd	200	1551
3	DSH	8	MN	Polymyopathy. No ocular signs.	↓↓	N	↑↑	↑	N	nd	1.037	180	3158
4	Siamese	8	MN	Forelimb stiffness then generalised polymyopathy. No ocular signs.	↓↓	N	↑↑	N	N	nd	1.018	180	3240
5	DSH	8	MN	Polymyopathy. No ocular signs.	↓↓	N	↑	N	N	nd	nd	nd	1634
6	DSH	10	MN	Polymyopathy. Increased retinal vessel tortuosity on fundic examination.	↓	N	↑↑	N	↑	N	nd	250	3712
7	DSH	12	FN	Polymyopathy. No ocular signs.	↓↓	N	↑↑	↑↑	↑	↓	1.038	190	8005
8	Burmese	13	FN	Mild dysphagia. Polymyopathy. No ocular signs.	↓↓	N	↑	N	N	nd	nd	170	13 850
9	DSH	9	MN	PU/PD. Polymyopathy. No ocular signs.	↓	N	↑↑	N	N	N	1.010	160	4464
10	DSH	6	MN	Polymyopathy. No ocular signs.	↓↓	N	↑	↑	↑	N	1.040	170	6230
11	Burmilla	10	FN	PU/PD/PP. Polymyopathy. No ocular signs.	↓	N	↑	↑	N	N	1.039	170	6480
12	DSH	12	MN	Polymyopathy. Bullous retinopathy on fundic examination.	↓↓	N	nd	↑	N	N	1.022	195	14 653
13	DSH	12	MN	PU/PD/PP. Polymyopathy. Bullous retinopathy on fundic examination.	↓↓	↑	↑	↑↑	N	↑	1.022	250	7800

^*DSH=domestic shorthair; FN=female neutered; MN=male neutered; PU=polyuria; PD=polydipsia; PP=polyphagia; K=potassium concentration; Na=sodium concentration; CK=creatine kinase concentration; Urea=urea concentration; Creat=creatinine concentration; TT₄ = total thyroxine concentration; USG=urine specific gravity; BP=systolic blood pressure; PACUSG=plasma aldosterone concentration;

↓ below reference range; ↓↓ far below reference range (≤2.5 mmol/l for K); ↑ above reference range; ↑↑ far above reference range (≥1000 iu/l for CK, ≥20 mmol/l for urea); N=normal; nd=not done.