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. Author manuscript; available in PMC: 2024 Dec 15.
Published in final edited form as: Neuropharmacology. 2023 Sep 30;241:109738. doi: 10.1016/j.neuropharm.2023.109738

Figure 2. Neonatal ethanol (EtOH) exposure did not significant affect neuronal densities in anterior thalamic nuclei at postnatal days 60–70.

Figure 2.

A) Sample images of neurons stained with anti-NeuN antibodies (red) and Venus+ GABAergic interneurons (green) from the Air (control) and EtOH groups (scale bars 100 μm). B) Summary graph illustrating the effect of third trimester-equivalent EtOH exposure on neuronal densities in the anterodorsal (AD), anteroventral (AV), or thalamic reticular (TRN) nuclei. Data were analysed by three-way ANOVA of log-transformed data. Posthoc analysis with the Holm-Šídák’s test yielded treatment p values ranging from >0.75 to >0.9 for both males and females in the AD nucleus, AV nucleus, and TRN. Control female, n=8 pups from 6 litters; EtOH female, n=7 pups from 4 litters; control male, n=6 pups from 3 litters; EtOH male, n=8 pups from 6 litters.