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. Author manuscript; available in PMC: 2024 Dec 15.
Published in final edited form as: Neuropharmacology. 2023 Sep 30;241:109738. doi: 10.1016/j.neuropharm.2023.109738

Figure 5. Neonatal ethanol (EtOH) exposure disrupted action potential firing in retrosplenial cortex-projecting anteroventral (AV) thalamic nucleus neurons at postnatal days 40–60.

Figure 5.

A) Representative traces of action potential firing evoked by current injection (600 pA) in AV nucleus neurons from the Air (control; black circles) and EtOH (red squares) groups. Whole trace scale bar = 50 ms X 10 mV; expanded trace scale bar = 5 ms X 10 mV. B) Mean effect of EtOH treatment on the instantaneous frequency of action potentials in females and males (data were analyzed by three-way ANOVA of aligned rank transformed data; Supplementary Data Tables). C) Mean effect of EtOH exposure on action potential amplitude in females and males (data were analyzed by three-way ANOVA of aligned rank transformed data; Supplementary Data Tables). Control female, n=7 pups from 5 litters; EtOH female, n=9 pups from 6 litters; control male, n=10 pups from 8 litters; EtOH male, n=10 pups from 5 litters.