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. Author manuscript; available in PMC: 2024 Dec 15.
Published in final edited form as: Neuropharmacology. 2023 Sep 30;241:109738. doi: 10.1016/j.neuropharm.2023.109738

Figure 7. Neonatal ethanol (EtOH) exposure did not affect spontaneous excitatory postsynaptic currents (sEPSCs) in retrosplenial cortex-projecting anteroventral (AV) thalamic nucleus neurons at postnatal days 40–60.

Figure 7.

A) Representative sEPSC traces from the Air (control) (black trace) and EtOH groups (red trace). Scale bar is 0.4 s X 40 pA. B) Mean effect of EtOH exposure on sEPSC frequency. C) Mean effect of EtOH exposure on sEPSC amplitude. D) Mean effect of EtOH exposure on sEPSC total charge. E) Mean effect of EtOH exposure on sEPSC rise time 10–90%. F) Mean effect of EtOH exposure on sEPSC decay time. Control female, n=9 pups from 6 litters; EtOH female, n=9 pups from 6 litters; control male, n=8 pups from 7 litters; EtOH male, n=10 pups from 6 litters.