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. 2023 Nov 27;38(2):340–350. doi: 10.1038/s41375-023-02093-7

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A UMAP plots of LN myeloid cell subclusters. Cells are shown separately for each clinical status (right panels). C1Q⁺ Mφ, complement component C1q positive macrophage; CD1C⁺ cDC2, CD1C-positive type2 conventional dendritic cell; Class Mono, classical monocyte; CLEC9A⁺ cDC1, CLEC9A-positive type1 cDC; Inter Mono, intermediate monocyte; LAMP3⁺ cDC, LAMP3-positive cDC; pDC, plasmacytoid DC. B Trajectory inference by Monocle2 for cDCs of TFHL, color-coded by cluster (left of the left panel) and pseudo-time (right of the left panel). Cells are shown separately for each cluster (right panel). C Comparison of proportions of each cluster in non-malignant MNCs per sample. The boxplots show the median (center line), interquartile range (box limits), minimum to max values (whiskers), and samples (dots) for each group. P values are shown only when there is a significant difference. *P < 5.0 × 10⁻².