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Schematic drawing of the impact of TRPA1 depletion or pore-function inhibition on FGR2c/PKCε signaling and tumorigenic outcomes. The depletion of TRPA1 expression by siRNA, but not its functional block using the antagonist A967079, significantly repressed the FGR2c/PKCε axis and the enhancement of EMT, as well as the MCL-1-dependent increase in cell invasion.
