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. 2024 Feb 8;6(2):e1048. doi: 10.1097/CCE.0000000000001048

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Copyright © 2024 The Authors. Published by Wolters Kluwer Health, Inc. on behalf of the Society of Critical Care Medicine.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 1. — Study population and main findings. A, Flowchart including the study population. B, Admission liver stiffness measurement of the right lobe (RLSM) accurately identified healthy control (HC) individuals (5.6 ± 2 kPa), acute liver failure (31.7 ± 17), and acute-on-chronic liver failure (76.3 ± 71 kPa) patients (Kruskall-Wallis). Acute liver failure (ALF) (n = 40), acute-on-chronic liver failure (ACLF) (n = 22), HC individuals (n = 9). C, Spleen size is increased in ACLF (14 ± 2.3 cm) compared with ALF (10.4 ± 2 cm) and HC individuals (9.5 ± 1.8 cm) (Kruskall-Wallis). III, Spleen stiffness is increased in ACLF (45.6 ± 26 kPa) compared with ALF (21 ± 16 kPa) and HC individuals (17.3 ± 10 kPa) (Kruskall-Wallis). D, Spleen stiffness is increased in ACLF (45.6 ± 26 kPa) compared with ALF (21 ± 16 kPa) and HC individuals (17.3 ± 10 kPa) (Kruskall-Wallis). *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001. ns = not statistically significant.