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. 2024 Jan 11;56(2):281–293. doi: 10.1038/s41588-023-01640-3

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 4 — a. Details of molecular categories with multiple category-defining alterations. The distribution on the UMAP plot according to fusion partners (KMT2Ar, NUP98r, ETS family, and APL categories) or mutation and fusions (NPM1 and GATA1 categories) are shown with colors representing the types of alterations. Age distributions according to fusion partners are also shown for KMT2Ar and NUP98r. Acute megakaryocytic/erythoid leukemia (AMKL/AEL) cases are shown separately in red. b. Proportion of molecular categories among different age groups (left: age<3, middle: 3<age<10, right: 10<age). Each column is colored according to the molecular categories, and categories associated with AMKL/AEL phenotypes are highlighted in a red square. Representative category names are shown in the columns.

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