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. 2024 Feb 13;81(1):88. doi: 10.1007/s00018-024-05125-2

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — A working model of Zbtb16-induced DADs occurrence and AF development by Txnip-Trx2 signaling in db/db mice. Zbtb16-Txnip-Trx2 pathway contributed to AF development in db/db mice by excess ROS generation-induced CaMKIIδ overactivation, abnormal SR Ca²⁺ release and DADs occurrence