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Model showing the potential effect of HSV-1 gG during infection. Infection of epithelial cells with HSV-1 (WT, left) or HSV-1 lacking gG expression (ΔgG, right) in the skin leads to the release of EVs. The EVs released by epithelial cells infected with HSV-1 WT contain higher levels of galectin-1 than those released upon HSV-1-ΔgG infection. Galectin-1 on EV induces neurite outgrowth, facilitating neuronal infection. Part of this figure was generated with Biorender.
