Fig. 2.
The mechanisms of selenomethionine (SeMet), arginine (Arg) and betulinic acid (BA) to attenuate T-2-induced immunotoxicity. GSH-Px = glutathione peroxidase; IL-1β = interleukin-1 beta; IL-6 = interleukin-6; MAPK = mitogen-activated protein kinase; MDA = malondialdehyde; Nrf2 = nuclear factor E2 related factor 2; ROS = reactive oxygen species; SOD = superoxide dismutase; T-2 = T-2 toxin; TNF-α = tumour necrosis factor alpha.