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. 2023 Aug 30;208(10):1088–1100. doi: 10.1164/rccm.202303-0491OC

Table 3.

IRRs of COPD Exacerbations and the Association with Glucagon-like Peptide 1 Receptor Agonists or Comparator Treatments, Inclusive of Recent Exacerbation* History

  Drug Exposure Clinical Model
Metabolic Model
Exploratory Analysis, Changes in HbA1c and BMI
IRR 95% CI P Value IRR 95% CI P Value IRR 95% CI P Value
Moderate and severe COPD exacerbations DPP-4is 1.22 0.90–1.65 0.21 1.13 0.83–1.55 0.44 2.01 1.00–4.15 0.057
SGLT2is 0.94 0.70–1.26 0.67 0.89 0.66–1.20 0.44 1.67 0.85–3.27 0.13
Sulfonylureas 1.59 1.25–2.03 0.0002 1.49 1.16–1.93 0.002 2.57 1.34–4.94 0.005
GLP-1RAs (ref)

Definition of abbreviations: BMI = body mass index; CI = confidence interval; COPD = chronic obstructive pulmonary disease; DPP-4i = dipeptidyl peptidase 4 inhibitor; GLP-1RA = glucagon-like peptide 1 receptor agonists; IRR = incidence rate ratio; ref = reference group; SGLT2i = sodium-glucose cotransporter 2 inhibitor.

*

Defined as ⩽12 months from index date.

The clinical model was adjusted for age; sex; race; Elixhauser Comorbidity Index; two-year health system encounter history; season of initiation; smoking history; number of exacerbations in the prior year; concurrent metformin use; and concurrent short-acting β-agonist, short-acting muscarinic antagonist, long-acting β-agonist, long-acting muscarinic antagonist, or triple-therapy use. The metabolic model was also adjusted for baseline HbA1c and baseline BMI.

To estimate the direct controlled effect, the exploratory analysis was also adjusted for change in BMI and change in HbA1c during the six-month study period.