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. 2024 Feb 21;16(1):2316575. doi: 10.1080/19490976.2024.2316575

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© 2024 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 8. — Therapeutic mechanisms of the engineered butyrate on IBD.

(a–c) The content of TNF-α, IL-6 and IL-1β in the serum of mice was measured. The serum was measured using the SenBeiJia Biological Technology mouse (TNF-α, IL-6 and IL-1β) ELISA Kit. (d–g) Relative expression level of TNF-α, IL-6, IL-1β and IL-10 in the colon. (H to I) The relative expression level of ZO1 and MUC2 in the colon. Data are means ± SEM (n = 6 to 8); *p < .05, **p < .01, ***p < .001 and ****p < .0001 by one-way ANOVA with Tukey’s multiple comparison test.