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. 2024 Feb 7;30(5):485–498. doi: 10.3748/wjg.v30.i5.485

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©The Author(s) 2024. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Farnesoid X receptor promoted proliferation of gastric cancer cells. A-F: Malignant proliferation assays, including cell viability (A and B), 5-ethynyl-2′-deoxyuridine (Edu) staining (C and D), andcolony formation assays (E and F), were performed in gastric cancer (GC) cells after transfection with the shNC or shFXR plasmid; G-L: Cell viability (G and H); Edu staining (I and J), andcolony formation assays (K and L) were performed in GC cells after transfection with the control or farnesoid X receptor-coding plasmid. Scale bar: 100 μm. ^aP < 0.05, ^bP < 0.01, ^cP < 0.001. These experiments were repeated three times. FXR: Farnesoid X receptor; Edu: 5-ethynyl-2′-deoxyuridine; NC: Negative control; NS: Not significant.