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. 2024 Jan 26;25(3):462–470. doi: 10.1038/s41590-023-01741-5

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 3 — Expression of CD16, NKG2D, Siglec-7, CD57, and PD-1 on CD3^-CD56⁺ NK cells. Participants were categorized into two subgroups, high response (HR, n = 4) and low response (LR, n = 4), based on whether that individual had an above or below average reduction in intact provirus⁺ cells with PG16-Db+3BNC117-Db. HIV^- participants were included for comparison (n = 3). Data reported as the percentage of cells that stained positive for the indicated marker (mean, SD). Correlations between percent reduction in intact copies and percentage of CD3^-CD56⁺ NK cells that stained positive for the indicated markers. Spearman’s r value shown.