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. 2024 Feb 20;15:1354297. doi: 10.3389/fimmu.2024.1354297

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Copyright © 2024 Masad, Idriss, Mohamed, Al-Sbiei, Bashir, Al-Marzooq, Altahrawi, Fernandez-Cabezudo and Al-Ramadi

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Therapeutic treatment with oral MH retards tumor growth in an IFNγ-dependent manner. Normal C57BL/6 (A) or IFNγ-deficient (B) mice were implanted with tumor cells and were orally gavaged daily starting on day 3 post implantation with 70% SC or 70% MH for 3 consecutive weeks. Each data point represents the mean ± SEM of the indicated mice within each experimental group, pooled from 2 independent experiments. Asterisks denote statistically significant differences between the SC-treated and MH-treated groups. p values were calculated using 2-way ANOVA, *** (p ≤ 0.001).