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. 2023 Nov 27;130(5):703–715. doi: 10.1038/s41416-023-02502-9

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The use of multiple drugs can target the inter-tumoural heterogeneity between patients, intra-tumoural heterogeneity within the same tumour (including the cancer stem cell subpopulation). Multiple drugs also target multiple pathways which may be redundant or compensatory allowing full cessation of proliferation, invasion and metastasis. Created using Biorender, May 2023.