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. 2024 Feb 20;49(1):58–64. doi: 10.1584/jpestics.D23-027

Fig. 2. Effect of dsRNA targeting β1 subunit on the sensitivity to imidacloprid of accommodated or non-accommodated cockroaches. (A) Schematic representation of the time course of the experiments. Cockroaches were exposed to either a sublethal dose of imidacloprid for 30 days (0.025 µg/day/cockroach; Accommodated cockroaches) or its solvent DMSO (Non-accommodated cockroaches). Then, each cockroach ingested 250 ng of either dsRNA-control or dsRNA-β1 (D30). An acute intoxication with imidacloprid at LD50 (2.5 µg/cockroach) was performed 96 hr after dsRNA ingestion (D34). (B) Mortality was assessed 48 hr after acute imidacloprid exposure (D36). Data are shown as the mean±S.E.M. (ns: non-significant, * p<0.05, ** p<0.01, nonparametric Mann–Whitney test; n=5 with 10 to 15 cockroaches per n).

Fig. 2. Effect of dsRNA targeting β1 subunit on the sensitivity to imidacloprid of accommodated or non-accommodated cockroaches. (A) Schematic representation of the time course of the experiments. Cockroaches were exposed to either a sublethal dose of imidacloprid for 30 days (0.025 µg/day/cockroach; Accommodated cockroaches) or its solvent DMSO (Non-accommodated cockroaches). Then, each cockroach ingested 250 ng of either dsRNA-control or dsRNA-β1 (D30). An acute intoxication with imidacloprid at LD50 (2.5 µg/cockroach) was performed 96 hr after dsRNA ingestion (D34). (B) Mortality was assessed 48 hr after acute imidacloprid exposure (D36). Data are shown as the mean±S.E.M. (ns: non-significant, * p<0.05, ** p<0.01, nonparametric Mann–Whitney test; n=5 with 10 to 15 cockroaches per n).