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. 2024 Jan 9;18(3):743–761. doi: 10.1002/1878-0261.13586

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© 2024 The Authors. Molecular Oncology published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 5 — Results of EV subpopulation analyses. The down‐sampling strategy was applied to select 20 000 EVs from pan‐cancer samples (n = 100) and 20 000 EVs from the control samples (n = 100). (A) Distribution of EV subpopulations between cancer and control groups. (B) Characteristic markers of cluster 1, cluster 3, and cluster 0 in (A). (C) Distribution of CD36 and ITGA1 between cancer and control groups in all clusters. The down‐sampling strategy was also applied to select 10 000 EVs from colorectal cancer samples (n = 20) and 10 000 EVs from the control samples (n = 100). (D) Distributions of EV subpopulations between colorectal cancer and control groups.